Titanium is ACTIVE promoting endothelial cells growth versus Stainless steel as a reference material

TITANIUM promotes endothelial cells growth by 100 to 400% in comparison with Stainless steel.
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Titanium-Nitride-Oxide is more ACTIVE than Titanium to reduce thrombocyte adhesion and fibrinogen adsorption

The blood compatibility of TITANIUM in part of thrombocyte adhesion and fibrinogen adsorption can be improved by the addition of Nitrogen (N) into the atomic external layer. Thrombocyte adhesion and fibrinogen adsorption are lower for TITANIUM-NITRIDE-OXIDE than for TITANIUM.
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Titanium-Nitride-Oxide is as ACTIVE as Sirolimus to reduce neointimal hyperplasia.

TITANIUM-NITRIDE-OXIDE is active like Sirolimus against neointimal hyperplasia in the porcine model.
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Stent coating with Titanium-Nitride-Oxide for reduction of neointimal hyperplasia in the porcine model

Titanium-Nitride-Oxide coating significantly reduces in-stent neointimal hyperplasia up to 47% demonstrating a true antiproliferative effect.
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Titanium is ACTIVE reducing platelet and fibrin deposition versus Carbon as a reference material

Despite the fact that both TITANIUM and CARBON are highly bio and haemo compatible, only TITANIUM does reduce platelet aggregation and fibrin deposition.
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Titanium unmatched blood compatibility over any other metallic substrate is ACTIVE reducing inflammation

TITANIUM offers the MOST biocompatible arterial interface MINIMIZING tissue reaction and toxic ions release thus reducing inflammation.
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